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Federal government websites often end in. The site is secure. The genetic component of Immunoglobulin-A IgA vasculitis is still far to be elucidated. To increase the current knowledge on the genetic component of this vasculitis we performed the first genome-wide association study GWAS on this condition. After quality control filters and GWAS imputation, patients and 1, controls remained in the datasets and were included in further analysis.

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Genetics of vasculitis.

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Arthritis Rheumatol. The course mirrors the actual Transat route, setting sail from Sainte-Adresse, to the north-west of Le Havre and ending in Martinique. Raychaudhuri S, et al. In addition, no consistent associations with IgA vasculitis susceptibility were detected outside the HLA region, probably due to an insufficient statistical power to detect risk variants with a moderate effect. Vasculitides constitute a heterogeneous group of diseases that often have overlapping clinical and pathological manifestations Consistent with the results obtained in a former study 5 , our data suggest the involvement of HLA class II region in the pathophysiology of IgA vasculitis, thus supporting the high relevance of the immune system in the development of this disease and suggesting that IgA vasculitis may be related to other class II vasculitides such as giant cell arteritis GCA 12 or AAV PLoS One. This study aimed at conducting the first GWAS of IgA vasculitis using the largest series of IgA vasculitis patients of European ancestry ever assessed for a genetic study. Miguel A. November 12, Because of that, further confirmatory studies in independent populations should be performed to validate our data. Rheum Dis Clin North Am. To confirm these results, we obtained direct genotypes of the Spanish cohort using a TaqMan probe for rs References 1. The genetic component of Immunoglobulin-A IgA vasculitis is still far to be elucidated.

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N Engl J Med. Arthritis Rheum. Genetically distinct subsets within ANCA-associated vasculitis. Arthritis Res Ther. Unlike the candidate gene approach, genome-wide association studies GWAS imply a hypothesis free analysis of hundreds of thousands of single-nucleotide polymorphisms SNPs across the whole genome 6. Outside the HLA, some potential signals located in different intronic and intergenic regions were observed Fig. The genetic component of Immunoglobulin-A IgA vasculitis is still far to be elucidated. To confirm these results, we obtained direct genotypes of the Spanish cohort using a TaqMan probe for rs Patients and Methods Study population A series of patients diagnosed with IgA vasculitis and 1, unaffected and unrelated controls were genotyped in this study. In addition, no consistent associations with IgA vasculitis susceptibility were detected outside the HLA region, probably due to an insufficient statistical power to detect risk variants with a moderate effect.